Preliminary studies reveal conolidine might inhibit unique ion channels, lowering neuronal excitability and limiting suffering signals. This system is especially related in neuropathic pain, in which abnormal signaling will cause persistent pain. Additionally, conolidine appears to impact G protein-coupled receptor (GPCR) pathways integral to discomfort notion. Computational docking r... https://julianq173ova7.wikirecognition.com/user